Actinomycetaceae lesions of the abdominal and anorectal area is quite rare. Their development is associated with acute perforation of internal organs (appendicitis, diverticulitis, crypts, various peptic diseases), surgical or other traumatic lesions, including lesions swallowed bone fragments or fish bones.
Another source of pelvic and abdominal actinomycotic infections have been identified.

The duration of the incubation period is unknown. It can vary within a wide range and up to several years (from time of infection to development of clinical forms of actinomycosis). The main clinical forms of actinomycosis: actinomycosis of the head, tongue and neck - torically actinomycosis abdominal - actinomycosis of the urinary organs - the lumpy skin - mycetoma (madorsky foot)- actinomycosis of the Central nervous system.

The incubation period lasts from 2 to 6 days but may last up to 17 days.
Acute herpetic gingivostomatitis usually affects children of preschool and younger school age, most often from 1 year to 3 years. In recent years, cases of disease of children 6 – 10 months., fed artificially with the first months of life.
Generalized form of herpes is possible in a baby born to a mother who does not have antibodies to herpes simplex virus. Infection such child who has not received from the mother's passive immunity leading to the development of severe septic disease with lesions of the serous membranes of the brain and internal organs.

The incubation period is 8 to 10 days.
Unlike other viral gastroenteritis enteric adenoviruses cause a longer disease (5 to 12 days, and sometimes up to 14 days).
The principal difference enteric adenoviruses from respiratory in that patients do not develop typical clinical signs, such as nasopharyngitis and keratoconjunctivitis, although viremia in these patients is also logged.
The disease is characterized by moderately severe intoxication, mild fever, persisting for several days. In those cases, when the disease lasts up to 2 weeks, patients often registered the fever is of the wrong type that is sometimes wavy character.

The tsetse fly inoculum of trypanosomes in the subcutaneous space by bloodsucking. Some parasites penetrate directly into the bloodstream, but most of them remained at the site of inoculation, where they multiply and cause a local education chancre. After the appearance of chancre, the trypanosomes are distributed in the interstitial spaces and lymphatic vessels and fall ultimately into the blood vessels, where they continue to multiply. Parasitemia is low, in the form of waves - each wave disappears at increasing the production of antibodies to surface glikoproteinom antigen of the parasite and comes back after 3 to 8 days as the formation of new antigenic variants.

The disease usually develops slowly and without symptoms of General intoxication, the body temperature is normal or subfebrile. In severe infestations, may have relatively high temperature and pulling or shadowassassin pain in the lower abdomen, worse during defecation. Appear painful tenesmus.
In severe colitis growing signs of intoxication, which is manifested by increase in temperature (usually wrong), decreased appetite, nausea, sometimes vomiting.

Pathogens of anisakidae man - larval stage of development of helminth genera: Anisakis, Contracaecum, Pseudoterranova, Hysterothylacium, relating to the order Ascaridida Skrjabin et Schikhobalova, 1940, the family Anisakidae Skrjabin et Korokhin, 1945,. The definitive hosts of these worms are many marine mammals (cetaceans, pinnipeds), predatory marine fish and fish-eating birds, in the gastro-intestinal tract which are parasitic males and females of anisakid.

The incubation period lasts from 7 to 16 days. The disease begins gradually. The body temperature rises, sometimes with chills, there is a weakness, headache, muscle pain, nausea, vomiting, anorexia. Fever increases and reaches 39-40°C. on examination revealed hyperemia of the face, neck, injection of vascular sclera. May be a small swollen lymph nodes. On 3-5th day of illness the patient's condition worsens, signs of dehydration (BP drops to 50-100 mmHg. calendar), oliguria increases.

Aspergillosis a variety of clinical manifestations that is determined by the immune status of the patient. In immunocompetent persons aspergillosis may be asymptomatic – in the form of a carriage, colonization, aspergilloma. With the deepening of immune disorders it can transform into an invasive form, which, depending on the degree of defects of immunity, is a chronic, subacute or acute, and the more pronounced immunological failure, the more acute nature of the disease.

Bagel very similar to syphilis, but not sexually transmitted. Most transmission occurs through dermal contact, or by sharing utensils.
Often called endemic syphilis, bejel characterized by the occurrence of skin and bone damage, he begins in the mouth and gradually progresses. The last stage is the most serious.
The incubation period from 2 weeks to 3 months.
Observed early and late stages of Bijela as in yaws and Pinta. The early stages include the incubation period, primary and secondary rashes.

The incubation period is 15 to 40 days, that is usually about 3 weeks, but may take up to 3 to 4 months.
In typical cases, the disease is dwuhfazno. In the first, acute phase, called Oroya fever, body temperature rises to 39 - 40 °C and remains at this level 10 - 30 days, then slowly decreases. The fever is accompanied by severe intoxication, fever, drenching sweat. There is a strong headache, bone, joint and muscle pain, malaise, insomnia, delirium or apathy, loss of appetite, nausea, vomiting. Appear on the skin hemorrhage, liver and spleen are enlarged, there may be jaundice.

Primarily affects the mucous membranes of the mouth and nose, ulcers occur with granulomatous bottom, slowly increasing in size, can be broken tongue, vocal cords, teeth fall out. The skin is involved in the process a second time (the process moves from mucous membranes), affected regional lymph nodes, the process can capture light (like tuberculosis), intestines, bones, and rarely the Central nervous system.